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Next time you are in a restaurant, consider ordering a curry. Eating curry regularly could save your life. Why? Because one of the ingredients of the humble curry is Turmeric. The golden coloured spice which belongs to the Ginger family, Turmeric comes from a plant called the 'Curcuma Longa' and contains a substance called Curcumin. Curcumin has been studied for the treatment of a number of diseases and disorders including Cancer, Hypertrophy, Skin Problems, Liver Disorders, Alzheimer's Disease, Stroke, Cystic Fibrosis, Aches, Pains, Pulmonary problems and many more. Curcumin has powerful anti-inflammatory and anti-oxidant properties and has been used in traditional Indian (Ayruvedic) and Chinese medicine for over three thousands years.
There have been many claims made about the
healing properties of Curcumin and there appears
to be a fairly healthy scientific rationale
behind this thinking. Curcumin is a COX-2
inhibitor and the future of pain relief is
thought to rely on COX-2 inhibition. COX-2 is an
enzyme responsible for inflammation and pain in
the body, therefore inhibition of COX-2 reduces
inflammation and pain. Medicines known as Non
Steroidal Anti Inflammatory Drugs (NSAIDS) work
on this principal. An example of an NSAID is
Ibuprofen.
Modern medicine is now looking to inflammation as the major theme underlying most major disease processes. As in headaches, it's the body's reaction to inflammation that maybe the cause of disorder and dysfunction.
Peter
Liu at the Canadian Institutes of Health
Research in Canada along with other researchers
studied the effects of Curcumin using a rat
model of cardiac hypertrophy (Enlarged Heart).
The study involved aortic banding which caused
pressure to the hearts of the rats which
resulted in hypertrophy (Enlarged Heart) and
eventual death. Curcumin was given in an oral
dose to the rats for a period of six weeks, this
was after two weeks of aortic banding. The
results showed a reversal of cardiac remodelling
(Remodelling is an alteration in the structure
or shape of the heart). Measures of
contractile dysfunction also returned close to
normal levels. The rats did not develop heart
failure.